PIA Publications
Neuroimaging
Articles of Interest
February 2017
Staubo, S., Aakre, J.,Vemuri, P., Syrjanen, J., Mielke, M., & Geda, Y. et al. (2017). Mediterranean diet, micronutrients and macronutrients, and MRI measures of cortical thickness. Alzheimer's & Dementia, 13(2), 168-177.
Corrada, M., Hayden, K., Bullain, S., Paganini-Hill, A., DeMoss, J., Aguirre, C., & Kawas, C. (2014). Age of Onset of Hypertension and Risk of Dementia in the Oldest-Old: The 90+ Study. Alzheimer's & Dementia, 10(4), P501.
December 2016
Dage, Jeffrey L. et al. "Levels of tau protein in plasma are associated with neurodegeneration and cognitive function in a population-based elderly cohort". Alzheimer's & Dementia 12.7 (2016): P877-P878. Web.
Bozoki, Andrea C., Monica Zdanukiewicz, and David C. Zhu. "The Effect Of Β-Amyloid Positivity On Cerebral Metabolism In Cognitively Normal Seniors". Alzheimer's & Dementia 12.12 (2016): 1250-1258.
Gispert, Juan Domingo et al. "Cerebrospinal Fluid Strem2 Levels Are Associated With Gray Matter Volume Increases And Reduced Diffusivity In Early Alzheimer's Disease". Alzheimer's & Dementia12.12 (2016): 1259-1272. Web.
Hanseeuw, Bernard J. et al. "Decreased Hippocampal Metabolism In High-Amyloid Mild Cognitive Impairment". Alzheimer's & Dementia 12.12 (2016): 1288-1296. Web.
August 2016
Gordon BA, Blazey T, Su Y, Fagan AM, Holtzman DM, Morris JC, Benzinger TL. Longitudinal β-Amyloid Deposition and Hippocampal Volume in Preclinical Alzheimer Disease and Suspected Non-Alzheimer Disease Pathophysiology. JAMA Neurol. 2016 Aug 22.
Mormino EC, Papp KV, Rentz DM, Schultz AP, LaPoint M, Amariglio R, Hanseeuw B, Marshall GA, Hedden T, Johnson KA, Sperling RA. Heterogeneity in Suspected Non-Alzheimer Disease Pathophysiology Among Clinically Normal Older Individuals. JAMA Neurol. 2016 Aug 22. doi: 10.1001/jamaneurol.2016.2237.
Power, M., Schneider, A., Wruck, L., Griswold, M., Coker, L., & Alonso, A. et al. (2016).Life-course blood pressure in relation to brain volumes. Alzheimer's & Dementia, 12(8), 890-899.
Clark, L., Racine, A., Koscik, R., Okonkwo, O., Engelman, C., & Carlsson, C. et al. (2016). Beta-amyloid and cognitive decline in late middle age: Findings from the Wisconsin Registry for Alzheimer's Prevention study. Alzheimer's & Dementia, 12(7), 805-814.
July 2016
Bourgeat P, Doré V, Savage G, Brown B, Laws S, Maruff P, Salvado O, Ames D, Martins RN, Masters CL, Rowe CC, Villemagne VL; AIBL Research Group. Clinical and cognitive trajectories in cognitively healthy elderly individuals with suspected non-Alzheimer's disease pathophysiology (SNAP) or Alzheimer's disease pathology: a longitudinal study. Lancet Neurol. 2016 Jul 19.
Cho H, Choi JY, Hwang MS, Lee JH, Kim YJ, Lee HM, Lyoo CH, Ryu YH, Lee MS. Tau PET in Alzheimer disease and mild cognitive impairment. Neurology. 2016 Jul 26;87(4):375-83.
JP, Schultz AP, Marshall GA, Boot B, Gomez-Isla T, Dumurgier J, LaPoint M, Scherzer C, Roe AD, Hyman BT, Sperling RA, Johnson KA. Temporal T807 binding correlates with CSF tau and phospho-tau in normal elderly. Neurology. 2016 Jul 29.
Burnham SC, Bourgeat P, Doré V, Savage G, Brown B, Laws S, Maruff P, Salvado O, Ames D, Martins RN, Masters CL, Rowe CC, Villemagne VL; AIBL Research Group. Clinical and cognitive trajectories in cognitively healthy elderly individuals with suspected non-Alzheimer's disease pathophysiology (SNAP) or Alzheimer's disease pathology: a longitudinal study. Lancet Neurol. 2016 Jul 19.
Chhatwal JP, Schultz AP, Marshall GA, Boot B, Gomez-Isla T, Dumurgier J, LaPoint M, Scherzer C, Roe AD, Hyman BT, Sperling RA, Johnson KA. Temporal T807 binding correlates with CSF tau and phospho-tau in normal elderly. Neurology. 2016 Jul 29.
Di Stefano, F., Kas, A., Habert, M., Decazes, P., Lamari, F., & Lista, S. et al. (2016). The phenotypical core of Alzheimer's disease-related and nonrelated variants of the corticobasal syndrome: A systematic clinical, neuropsychological, imaging, and biomarker study. Alzheimer's & Dementia, 12(7), 786-795.
Buckley, R., Maruff, P., Ames, D., Bourgeat, P., Martins, R., & Masters, C. et al. (2016). Subjective memory decline predicts greater rates of clinical progression in preclinical Alzheimer's disease.Alzheimer's & Dementia, 12(7), 796-804.
Clark, L., Racine, A., Koscik, R., Okonkwo, O., Engelman, C., & Carlsson, C. et al. (2016). Beta-amyloid and cognitive decline in late middle age: Findings from the Wisconsin Registry for Alzheimer's Prevention study. Alzheimer's & Dementia, 12(7), 805-814.
June 2016
Lockhart SN, Baker SL, Okamura N, Furukawa K, Ishiki A, Furumoto S, Tashiro M, Yanai K, Arai H, Kudo Y, Harada R, Tomita N, Hiraoka K, Watanuki S, Jagust WJ. Dynamic PET Measures of Tau Accumulation in Cognitively Normal Older Adults and Alzheimer's Disease Patients Measured Using [18F] THK-5351. PLoS One. 2016 Jun 29.
September 2015
Schmidt, M., Chiao, P., Klein, G., Matthews, D., Thurfjell, L., & Cole, P. et al. (2015). The influence of biological and technical factors on quantitative analysis of amyloid PET: Points to consider and recommendations for controlling variability in longitudinal data. Alzheimer's & Dementia, 11(9), 1050-1068.
Varley, J., Brooks, D., & Edison, P. (2015). Imaging neuroinflammation in Alzheimer's disease and other dementias: Recent advances and future directions. Alzheimer's & Dementia, 11(9), 1110-1120.
Witte, M., Foster, N., Fleisher, A., Williams, M., Quaid, K., & Wasserman, M. et al. (2015). Clinical use of amyloid-positron emission tomography neuroimaging: Practical and bioethical considerations. Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring, 1(3), 358-367.
August 2015
Sabri, Osama et al. Florbetaben PET Imaging To Detect Amyloid Beta Plaques In Alzheimer's Disease: Phase 3 Study. Alzheimer's & Dementia 11.8 (2015): 964-974.
Thal, Dietmar Rudolf et al. [18F]Flutemetamol Amyloid Positron Emission Tomography In Preclinical And Symptomatic Alzheimer's Disease: Specific Detection Of Advanced Phases Of Amyloid-Β Pathology. Alzheimer's & Dementia 11.8 (2015): 975-985.
Hake, Ann et al. Florbetapir Positron Emission Tomography And Cerebrospinal Fluid Biomarkers. Alzheimer's & Dementia 11.8 (2015): 986-993.
Sabbagh, Marwan et al. Florbetapir PET, FDG-PET And MRI In People With Down Syndrome With And Without Symptomatic Alzheimer's Disease. Alzheimer's & Dementia 9.4 (2013): P290.
June 2015
Teipel, S., Bruno, D., Grothe, M., Nierenberg, J., & Pomara, N. (2015). Hippocampus and basal forebrain volumes modulate effects of anticholinergic treatment on delayed recall in healthy older adults. Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring, 1(2), 216-219.
Doherty, B., Oh, J., Schultz, S., Koscik, R., Dowling, N., & Barnhart, T. et al. (2014). Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimer's & Dementia, 10(4), P66.
Hwang, K., Lazaris, A., Eastman, J., Teng, E., Thompson, P., & Gylys, K. et al. (2015). Plasma BDNF levels associate with Pittsburgh Compound B binding in the brain. Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring, 1(2), 187-193.
- Alliance of Women Alzheimer's Researchers (AWARE)
- Atypical Alzheimer's disease and Associated Syndromes
- Biofluid Based Biomarkers
- Cognition
- Clinical Trials Advancement and Methods
- Design and Data Analytics
- Diversity and Disparities
- Down Syndrome and Alzheimer's Disease
- Electrophysiology
- Immunity and Neurodegeneration
- Neuroimaging
- Neuropsychiatric Syndromes
- Non-pharmacological Interventions
- Nutrition, Metabolism, and Dementia
- Perioperative Cognition and Delirium
- Reserve, Resilience and Protective Factors
- Subjective Cognitive Decline
- Technology
- Vascular Cognitive Disorders
- Alliance of Women Alzheimer's Researchers (AWARE)
- Atypical Alzheimer's disease and Associated Syndromes
- Biofluid Based Biomarkers
- Cognition
- Clinical Trials Advancement and Methods
- Design and Data Analytics
- Diversity and Disparities
- Down Syndrome and Alzheimer's Disease
- Electrophysiology
- Immunity and Neurodegeneration
- Neuroimaging
- Neuropsychiatric Syndromes
- Non-pharmacological Interventions
- Nutrition, Metabolism, and Dementia
- Perioperative Cognition and Delirium
- Reserve, Resilience and Protective Factors
- Subjective Cognitive Decline
- Technology
- Vascular Cognitive Disorders
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